MOGES
Morpho-functional
(D) Dilated
(H) Hypertrophic
(H(Obs)) Hypertrophic Obstructive
(H(noObs)) Hypertrophic non Obstructive
(R) Restrictive
(A) ARVC
(NC) LVNC
(NS) Nonspecific phenotype
(NA) Information not available
(0) Unaffected
(E[D]) Early Diagnosis of Dilated
(E[H]) Early Diagnosis of Hypertrophic
(E[R]) Early Diagnosis of Restrictive
(E[A]) Early Diagnosis of ARCV
(R EMF) Endomyocardial Fibrosis
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Organ/system involvement
(H) Heart
(M) Muscle, Skeletal
(N) Nervous
(C) Cutaneous
(E) Eye
(A) Auditory
(K) Kidney
(G) Gastrointestinal
(S) Skeletal
(Lu) Lung
(Li) Liver
(0) Absence of organ/system involvement
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Genetic
(N) Family History Negative
(U) Family History Unknown
(AD) Autosomal Dominant
(AR) Autosomal Recessive
(XLR) X-linked Recessive
(XLD) X-linked Dominant
(XL) X-linked
(M) Matrilineal
(0)
(Undet) Inheritance Still Undetermined
(S) Phenotipically Sporadic
Etiological Annotation
(G) Genetic etiology
(G-OC) Obligate Carrier
(G-ONC) Obligate Non-Carrier
(G-DN) De novo
(G-Neg) Genetic test negative
(G-N) Genetic defect Not Identified
(G-A) Genetic Amiloydosis
(0) No genetic test
(M) Myocarditis
(V) Viral infection
(AI) Autoimmune/immune-mediate
(AI-S) Autoimmune/immune-mediate Suspected
(AI-P) Autoimmune/immune-mediate Proven
(A) Amyloidosis
(I) Infectious, non viral
(T) Toxicity
(Eo) Hypereosinophilic heart disease
(A-K) Amyloidosis type K
(A-L) Amyloidosis type L
(A-SAA) Amyloidosis type SAA
(Other)
Description
Gene
Mutation
Color Code
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Stage
ACC-AHA stage
A
B
C
D
NA
NU
NYHA class
I
II
III
IV
M
0
O
0
G
0
E
0
S
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